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Research Resources:
Alopecia, hair loss abstracts - Dream Pharmaceuticals, Rx online
Med Oral. 2002 Jul-Oct;7(4):303-8.
Alopecia areata of dental origin.
[Article in English, Spanish]
Gil Montoya JA, Cutando Soriano A, Jimenez Prat J.
Facultad de Odontologia Paseo de la Cartuja s/n 18071 GRANADA, Spain. jagigr.es
The association of alopecia areata and infectious foci of dental origin is relatively common, and may be explained by the autoimmune nature of the disorder. We describe a case of alopecia areata with no apparent cause and that was effectively resolved by eliminating a focalized dental infection via endodontic treatment. The presence of common immune mediators in the pathogenesis of both alopecia areata and dental infection could account for the dental origin of the hair loss. In this sense, patients with localized alopecia should be subjected to careful exploration of the oral cavity in search of possible dental infections.
online pharmacy ref. source: www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=12134132&dopt=Abstract
J Craniofac Surg. 2002 Jul;13(4):497-500.
Complete osseous regeneration of a large skull defect in a patient with cutis aplasia: a conservative approach.
Rhee ST, Colville C, Buchman SR, Muraszko K.
Section of Plastic Surgery, University of Michigan Medical Center, Ann Arbor, Michigan 48109-2109, USA.
Cutis aplasia (or aplasia cutis congenita) is a congenital absence of all skin layers, often extending through bone. This defect usually occurs in the scalp and can be extensive, exposing the dura mater, and deeper meninges. Treatment regimens for cutis aplasia have included early operative intervention, including skin and bone grafts, local scalp flaps, or free flaps to close the defect. In addition to the significant perioperative risks, these invasive procedures may inhibit the osteogenic potential of the dura to initiate and sustain bony closure of the defect. We report a case of an infant with Adams-Oliver syndrome and cutis aplasia involving a large portion of the skull that was treated conservatively with topical Silvadene dressings. No surgical treatment of bone or soft tissue reconstruction was necessary. This case report is the first to our knowledge to document complete bony restoration of the cranial vault through serial three-dimensional CT scans. The intensive therapeutic intervention in this case report allowed early discharge from the hospital, a gradual amelioration of the patient's alopecia as the hair-bearing scalp slowly covered the defect, and precluded the need for any subsequent bony reconstruction of the cranial vault. We hypothesize that conservative treatment of cutis aplasia maintains dural induction of osseous regeneration, and any treatment plan for bony defects of cutis aplasia should consider maintenance of dural integrity. Although further investigation is warranted, an initial trial of antimicrobial dressing care might optimally promote secondary closure of the cranial vault without the need for surgical intervention.
online pharmacy ref. source: www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=12140410&dopt=Abstract
Am J Dermatopathol. 2002 Jun;24(3):204-8.
An investigation of apoptosis in androgenetic alopecia.
Cowper SE, Rosenberg AS, Morgan MB.
Departments of Dermatology, University of South Florida, Tampa, FL, USA. shawn.cowpeale.edu
While the androgens, including dihydrotestosterone (DHT), have been implicated in the development of androgenetic alopecia (AGA), the exact mechanism by which they exert their effects is unknown. As apoptosis is an integral component of the normal cycling of human hair, we investigated individuals clinically affected by AGA to assess whether objective differences in the expression of apoptosis related immunohistochemical markers could be observed in scalp biopsies. Specimens from 13 alopecic male cadavers were stained with bcl-2 and terminal deoxynucleotidetransferase dUTP fluorescein nick end-labeling (TUNEL) methods to assess apoptotic activity in affected and unaffected areas of the scalp. Immunoreactivity was analyzed by quantifying nuclear staining differences within the same individual. Sections from two living human volunteers were obtained to establish the method validity. Significant differences in bcl-2 expression were observed between areas of the scalp clinically affected and unaffected by AGA. The Gaussian distribution of bcl-2 staining suggests that a relatively uniform population of follicles exists at the frontal hairline and/or synchrony of follicular cycling occurs in AGA. The apoptosis "hot spot" described by TUNEL staining in the bulge-isthmus region of the murine follicle is also identifiable in the human follicle.
online pharmacy ref. source: www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=12140435&dopt=Abstract
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