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Am J Ophthalmol. 2001 Jan;131(1):55-60.
Perimetric sensitivity and retinal thickness in eyes with macular edema resulting from branch retinal vein occlusion.
Imasawa M, Iijima H, Morimoto T.
Department of Ophthalmology, Yamanashi Medical University, Tamaho, Yamanashi, Japan.
PURPOSE: To document and study the relationship between perimetric sensitivity and retinal thickness at the foveal and parafoveal points in eyes with macular edema associated with branch retinal vein occlusion. METHODS: A prospective study was performed using the central 10-2 Humphrey perimetry program and optical coherence tomography. Seventeen eyes with branch retinal vein occlusion were eligible showing macular edema without macular nonperfusion or massive retinal hemorrhage. RESULTS: The sensitivity and retinal thickness were significantly correlated at the fovea (r = -0.629, P =.006) as well as 1 (r = -0.656, P <.0001) and 3 (r = -0.885, P <.0001) degrees apart from the fovea. The visual acuity that is expressed as a logarithm of the minimum angle of resolution (LogMAR) also correlated with retinal thickness (r = 0.591, P =.011). CONCLUSION: The increased retinal thickness resulting from macular edema is closely correlated with retinal sensitivity as measured by automated static perimetry both at the fovea and parafovea. Measuring the retinal thickness using optical coherence tomography may be useful in monitoring macular edema in eyes with branch retinal vein occlusion.
Online source: www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=11162980&dopt=Abstract hemorrhage
Am J Ophthalmol. 2001 Jan;131(1):138-40.
Solitary nonreactive choroidal tuberculoma in a patient with acquired immune deficiency syndrome.
DiLoreto DA Jr, Rao NA.
Department of Ophthalmology, University of Southern California School of Medicine (D.A.D.), Los Angeles, CA, USA.
PURPOSE: To report a case of a solitary, nonreactive choroidal tuberculoma in a patient with acquired immune deficiency syndrome (AIDS). METHOD: Case Report. RESULTS: A 26-year-old male patient with AIDS and systemic tuberculosis was found to have a solitary 1.5-disc-diameter elevated mass just superior and temporal to the optic disc. There was no associated inflammation, exudate, hemorrhage, or serous retinal detachment. Fluorescein angiography showed late hyperfluorescence in a staining pattern. The mass quickly regressed with antituberculosis therapy. CONCLUSIONS: Choroidal tuberculoma can present with little associated inflammation or retinal change in a patient with AIDS. The clinical history and knowledge of opportunistic choroidal infections in patients with AIDS helps to make the diagnosis.
Online source: www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=11162995&dopt=Abstract hemorrhage
Arch Soc Esp Oftalmol. 2003 Feb;78(2):103-6.
[Cryptococcal choroiditis in a patient with acquired immunodeficiency syndrome]
[Article in Spanish]
Fernandez Gonzalez MC, Perez Blazquez E, Galvez Ruiz A, Bonales Daimiel JA.
Servicio de Oftalmologia, Hospital 12 de Octubre, Madrid, Espana.
PURPOSE/METHODS: We report the case of a 31 year old male with presumed cryptococcal choroiditis associated with cryptococcal meningitis and AIDS. Multiple yellowish-whitish small choroidal lesions, retinal hemorrhages and cotton-wool spots appeared in the posterior pole associated with vision loss. We also carried out differential diagnosis with other frequent diseases and a review of the treatments. RESULTS/CONCLUSIONS: Cryptococcus neoformans is an opportunistic fungus with a predilection for infecting the meninges in patients with Acquired Immunodeficiency Syndrome AIDS (10%). Ocular manifestation usually includes cranial nerve palsies or papilledema secondary to increased intracranial pressure. Cryptococcal choroiditis is less frequent (5% of patients with meningitis) (Arch Soc Esp Oftalmol 2003; 78: 103-106).
Online source: www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=12647251&dopt=Abstract hemorrhage [PubMed - in process]
Jpn J Ophthalmol. 2001 Jan-Feb;45(1):108-10.
Branch retinal vein occlusion in the right eye and retinal hemorrhage in the left in a patient with classical Tsutsugamushi disease.
Nagaki Y, Hayasaka S, Kadoi C, Matsumoto M, Sakagami T.
Department of Ophthalmology, Toyama Medical and Pharmaceutical University, Toyama, Japan.
PURPOSE: To report branch retinal vein occlusion and retinal hemorrhages associated with tsutsugamushi disease. METHODS: Case report of a 60-year-old woman who complained of fever, chills, headache, lymphadenopathy, and blurred vision in the right eye following an insect bite to the lower right forehead. RESULTS: Serological findings showed elevated titers for the strains of Rickettsia tsutsugamushi. Ophthalmologic examination disclosed bilateral conjunctival injection, flame-shaped hemorrhage in her right fundus, and scattered hemorrhage in her left fundus. Fluorescein angiography demonstrated dye leakage and dilation of capillaries. CONCLUSIONS: Branch retinal vein occlusion associated with classical tsutsugamushi disease, as demonstrated in our patient, may be rare.
Online source: www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=11163055&dopt=Abstract hemorrhage
Free Radic Biol Med. 2001 Jan 15;30(2):198-207.
Salicylate and cocaine: interactive toxicity during chicken mid-embryogenesis.
Venturini L, Sparber SB.
Department of Pharmacology, University of Minnesota, Minneapolis, MN 55455-0217, USA.
Increased free radical production, due to ischemia and reperfusion, has been postulated as a cause of cocaine's (COC) developmental toxicity. Salicylate reacts with hydroxyl free radicals (*OH) to form stable, quantifiable reaction products, which can be measured with high-pressure liquid chromatography (HPLC). To determine if chicken embryos' brains and hearts were exposed to increased *OH concentrations after injection of COC, an injection of a nontoxic dose of sodium salicylate (NaSAL, 100 mg/kg egg, or 5 mg/egg), followed by 5 injections of COC (13.5 mg/kg or 0.675 mg/egg, every 1.5 h), was administered to eggs containing embryos on the 12th day of embryogenesis (E12). In addition to finding increased *OH concentrations in E12 embryonic hearts and brains, we observed that the developmental toxicity of COC, manifest as vascular disruption (hemorrhage) and lethality, was enhanced by NaSAL injection. These results confirm and extend results of similar experiments performed upon older embryos (E18), and indicate that increased &z.rad;OH concentration in embryonic tissues after COC exposure and toxic interactions of COC and NaSAL can also occur at an earlier stage of development. The results are discussed in light of possible exposure of human fetuses to both COC and salicylates, since COC-abusing pregnant women can be misdiagnosed with pre-eclampsia and aspirin is used to treat this syndrome.
Online source: www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=11163537&dopt=Abstract hemorrhage
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