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Ann Fr Anesth Reanim. 2002 Oct;21(8):668-71.
[Chest pain and sulprostone during postpartum hemorrhage]
[Article in French]
Bayoumeu F, Aallali M, Koebele A, Steschenko G, Laxenaire MC.
Service d'anesthesie-reanimation, maternite regionale, 10, rue du Dr Heydenreich, 54042 Nancy, France. secretariat.anesthesiaternite.chu-nancy-fr
A case of chest pain in a 31-year-old woman after vaginal delivery with epidural analgesia during sulprostone administration is described. Chest pain occurred shortly after sulprostone was started and disappeared when sulprostone was stopped. Ischaemia related data were negative. Angiographically coronary arteries were normal. Coronary artery spasm aetiology was retained. Sulprostone pharmacology is summarized. Coronary artery effects are compared with literature reports. Recommendations before sulprostone use are underlined.
Online source: www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=12471787&dopt=Abstract hemorrhage
Am J Pathol. 2002 May;160(5):1755-65.
Increased mortality and inflammation in tumor necrosis factor-stimulated gene-14 transgenic mice after ischemia and reperfusion injury.
Souza DG, Soares AC, Pinho V, Torloni H, Reis LF, Teixeira MM, Dias AA, Martins MT.
Department of Biochemistry and Immunology, Universidade Federal de Minas Gerais, Belo Horizonte, Brazil.
TSG-14/PTX3 is a gene inducible by tumor necrosis factor (TNF)-alpha, interleukin-1 beta, and lipopolysaccharide in fibroblasts, macrophages, and endothelial cells. It encodes a 42-kd secreted glycoprotein that belongs to the pentraxin family of acute-phase proteins. Recently, we demonstrated that TSG-14 transgenic mice (TSG-14tg) overexpressing the murine TSG-14 gene under control of its own promoter are more resistant to lipopolysaccharide-induced shock and to polymicrobial sepsis caused by cecal ligation and puncture. Here we show that after ischemia and reperfusion (I/R) injury, TSG-14tg mice have an impaired survival rate, which appeared secondary to a markedly increased inflammatory response, as assessed by the local (duodenum and ileum) and remote (lung) enhancement in vascular permeability, hemorrhage, and neutrophil accumulation. Moreover, tissue concentrations of TNF-alpha, interleukin-1 beta, KC, and MCP-1 were higher in TSG-14tg as compared to wild-type mice after I/R injury. Of note, elevated TNF-alpha concentrations in serum were only observed in TSG-14tg mice and blockage of TNF-alpha action prevented lethality of TSG-14tg mice. These results demonstrate that transgenic expression of TSG-14 induces an enhanced local and systemic injury and TNF-alpha-dependent lethality after I/R. Taken together, our data point to a critical role of TSG-14 in controlling acute inflammatory response in part via the modulation of TNF-alpha expression.
Online source: www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=12000727&dopt=Abstract hemorrhage
Can Vet J. 1999 Aug;40(8):571-6.
Severe renal hemorrhage caused by pyelonephritis in 7 horses: clinical and ultrasonographic evaluation.
Kisthardt KK, Schumacher J, Finn-Bodner ST, Carson-Dunkerley S, Williams MA.
Department of Large Animal Surgery and Medicine, Auburn University, Auburn, Alabama 36849-5522, USA.
Case records of 7 horses diagnosed with pyelonephritis were reviewed to determine common features that might aid in diagnosis, treatment, and prognosis of this disease. All 7 horses had been admitted for evaluation of hematuria. During cystoscopy of 5 horses, hemorrhage was observed from one or both ureters. Renal biopsy of 1 horse, laboratory analysis of ureteral discharge of 2 horses, and renal ultrasonography of all horses indicated that pyelonephritis was the cause of hemorrhage. Sonographic renal changes included decreased length, increased echogenicity, abnormal outline, loss of corticomedullary distinction, pyelectasia, and focal hypoechoic or hyperechoic cortical defects. Renal hemorrhage in all horses eventually resolved but recurred in 4 of 5 horses that were followed long-term.
Online source: www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=12001337&dopt=Abstract hemorrhage
Scand J Urol Nephrol. 2002 Feb;36(1):65-70.
Pentoxifylline attenuates reperfusion injury in testicular torsion.
Savas C, Dindar H, Bilgehan A, Ataoglu O, Yucesan S.
Department of Pediatric Surgery, Suleyman Demirel University Medical School, Isparta, Turkey. savar.net
OBJECTIVES: An experimental study was designed to evaluate the effects of pentoxifylline (Ptx) on lipid peroxidation, and histopathology in both testes after unilateral testicular torsion and detorsion. MATERIALS AND METHODS: Forty adult male albino Wistar rats were randomly divided into 4 groups of sham operation, sham operation with Ptx, torsion and detorsion, torsion and detorsion with Ptx. After intraperitoneal administration of Ptx at a dose of 50 mg/kg 15 min before torsion; right testes of the rats underwent 30 min of torsion and 30 min of detorsion. Malondialdehyde (MDA) levels were assayed and histopathological changes were evaluated in both testes of all groups. RESULTS: Unilateral testicular torsion and detorsion caused an increase in the MDA levels of both testes. Histopathological evaluation showed interstitial hemorrhage on the ipsilateral side. Pentoxifylline decreased MDA levels on both side, and attenuated interstitial injury on the ipsilateral side. CONCLUSIONS: The results of this study suggest that pentoxifylline treatment attenuates reperfusion damage on both side, possibly with its effects on blood flow and neutrophils. However, further studies are necessary to evaluate the effects of pentoxifylline on testicular torsion.
Online source: www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=12002361&dopt=Abstract hemorrhage
Cranio. 2002 Apr;20(2):116-24.
Microscopic analysis of the temporomandibular joint in rabbits (Oryctolagus cuniculus L.) using an occlusal interference.
Chaves K, Munerato MC, Ligocki A, Lauxen I, de Quadros OF.
Department of Conservative Dentistry, UFRGS, Porto Alegre, RS, Brazil. kchaveaz.com.br
The purpose of this study was to assess the tissue alterations in the temporomandibular joint (TMJ) of the New Zealand White rabbit (Oryctolagus cuniculus L.), after a unilateral occlusal interference insertion on the animal's right side back teeth. A total of 36 animals were used, thirty of which belonged to the experimental group and six to the control group. We established three experimental periods: 24 hours, three days and seven days. The control group animals were divided two by two; each pair followed the same experimental periods of the former one. The experimental group animals were submitted to the use of a 0.3 mm thick metallic cap with a visor. All animals were euthanized, and the TMJs were removed. Using a microscope for examination we observed, in all experimental periods, the presence of intra-articular hemorrhage in the supra- and infra-disk compartments as well as in the retro-disk zone. There were no inflammatory cells detected. The thickness of the condylar fibrocartilage presented significant alterations among the animals of the three experimental groups. In the left TMJs no inflammatory cells were detected. The results suggest that the insertion of a unilateral occlusal interference in rabbit back teeth does not cause any inflammatory intra-articular process within seven days; however, it does cause bilateral intra-articular hemorrhage and a larger compression of the condylar fibrocartilage in the joint opposite the side where the interference is placed. We also concluded that, in order to do research on the temporomandibular joint using animals, it is necessary have an independent (or separate) group of animals as controls.
Online source: www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=12002827&dopt=Abstract hemorrhage
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